この研究は、成獣のSprague-Dawleyラットに、通常の携帯電話からの電磁放射への1日6時間、126日間(18週間)の亜慢性ばく露を行い、いくつかの損傷関連タンパク質のmRNA合成のレベルの変化をRT-PCR法で評価した。カルシウムATPase、神経細胞接着分子、神経成長因子、血管内皮成長因子を評価した。その結果、ばく露を受けたラットの脳において、これらのタンパク質のmRNAの有意な増加が観察された、と報告している。
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To check the hypothesis that cell phone irradiation can cause cellular damage to the rat brain leading to increased mRNA transcription of injury-associated proteins.
Fourteen 3-month-old male adult rats, weighing 250 to 350 g, were used.
周波数 | 1.9 GHz |
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タイプ |
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ばく露時間 | repeated daily exposure, 2 x 3 h/day, for 18 weeks |
Modulation type | unspecified |
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ばく露の発生源/構造 |
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Distance between exposed object and exposure source | 1 cm |
チャンバの詳細 | The rats were placed in holding units consisting of ventilated 5.1 cm x 15.2 cm PVC tubes fitted with 0.59-liter clear plastic bottle tops on one end and common 7.6-cm bolts with nuts at the other end. As the rats grew larger, new units were made using 8.9-cm PVC tubes, 1-liter bottles, and 10.2-cm bolts with nuts. |
ばく露装置の詳細 | Each cell phone was positioned 1 cm from the head of the rats, at equal distances between two rats in holding chambers. |
Sham exposure | A sham exposure was conducted. |
Additional information | Rats were exposed for 3 hours, followed by a 30-minute rest period outside of the tubes and a second exposure for 3 more hours per day. |
A statistically significant mRNA up-regulation of all investigated injury-associated proteins in the brains of rats exposed to cell phone irradiation was found. The differences in temperature measured were not statistically sinificant.
The authors conclude that relative chronic exposure to cell phone microwave irradiation may result in cumulative injuries that could eventually lead to clinically significant neurological damage.
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