To replicate the effect of low-intensity millimeter waves of previous studies on isolated nerve function and to characterize its dependence on irradiation intensity. Exposures were accompanied by a high-rate electrical stimulation of the nerve.
| Exposure | Parameters |
|---|---|
|
Exposure 1:
41.34 GHz
Exposure duration:
2 HRS trials of 23 min separated by an interval of 53 min
|
|
| Frequency | 41.34 GHz |
|---|---|
| Type | |
| Exposure duration | 2 HRS trials of 23 min separated by an interval of 53 min |
| Exposure source | |
|---|---|
| Distance between exposed object and exposure source | 52 mm |
| Chamber | exposure bath maintained at 11 to 12°C filled with minerial oil and equipped with two pairs of saline bridge electrodes |
| Sham exposure | A sham exposure was conducted. |
| Additional info | first HRS trial without millimeter wave exposure, the second one with millimeter wave exposure or sham exposure |
| Measurand | Value | Type | Method | Mass | Remarks |
|---|---|---|---|---|---|
| power density | 0.02 mW/cm² | - | measured | - | - |
| power density | 0.1 mW/cm² | - | measured | - | - |
| power density | 0.5 mW/cm² | - | measured | - | - |
| power density | 2.6 mW/cm² | - | measured | - | - |
Millimeter waves had no effect on the conditioning compound action potentials, but significantly attenuated the high-rate electrical stimulation-caused decrease of the test compound action potentials (conditioning/test compound action potential (CAP), i.e. the CAPs evoked by the first and the second stimuli in a pair, respectively). The magnitude of this effect was virtually the same at field intensities of 0.02, 0.1, and 2.6 mW/cm². Exposure at 0.5 mW/cm², however, did not cause statistically significant changes.
The findings are consistent with earlier observations of this millimeter wave effect and provide further evidence for its nonthermal mechanism.
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